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LUMBAR REPOSITIONING DEFICIT IN A SPECIFIC LOW BACK PAIN POPULATION.
  Spine, Vol. 28, No. 10, May 15, 2003.

            Peter B. O’Sullivan, PhD et al

 

ABSTRACT

Proprioception is the sensation of position and movement at the joints in the body, along with an understanding of the degrees of force and effort of muscular contraction, and even the timing of the contraction.  Injury to peripheral joints have been shown to cause reductions in position sense and proprioception, and this may cause abnormal loading across the joint, resulting in pain and damage to the articular surfaces.  While some studies have indicated that persons with chronic LBP have deficits to lumbar spine position sense, others have not.  This study sought to determine if persons with a specific spinal instability syndrome (LSI, or lumbar spine instability, flexion pattern) have alterations in their ability to reproduce a pelvic neutral position when compared to an age/sex/weight matched normal control group.

 

Subjects with LSI, a total of 15, were carefully chosen by experienced manual therapists.  Exclusion criteria included:  past history of training in motor control, recent back surgery, neurological involvement, and pain that prohibited them from performing the test.  All had a history of low back pain for at least 3 months, and a confirmed diagnosis of LSI flexion pattern.  This was defined as a history of chronic/recurrent LBP secondary to a flexion injury, with trivial movements related to flexion, flexion/rotation, and sustained flexion causing symptoms.  These subjects had full range of movement with pain going into or returning from flexion, loss of segmental lordosis at the involved level, increases segmental mobility at the involved level, and difficulty assuming and maintaining a neutral lordotic curve.  The control group consisted of 15 subjects who were age, sex, height and weight matched, with no history of LBP.

 

The patients were seated, hips and knees at 90 degrees, with surface sensors placed at T12, L2, L4, and S2.  The device used to measure spinal position was a 3-Space Fastrack Model 3SF00002.  This device uses an external recorder that is able to measure the space and location of the electrodes.  Patients were placed into a pelvic neutral position, which was then recorded as a baseline.  They assumed a fully flexed position while seated, and then had to return to the neutral position for a total of 5 trials.  Each seated flexions stretch was held for 5 seconds.

 

Results showed that the patients with LSI had significant reductions in the ability to return to the previously chosen neutral position.  This indicates that there may be deficits in both motor control of specific muscles, along with reduced coordination between muscle groups.  It has already been determined that unisegmental muscles in the spine have 2-6 times the density of muscle spindles that polysegmental muscles have.  The unisegmental muscles may act to control specific segments, but most likely also work in tandem with the polysegmental muscles.  The data from this study found that no specific level revealed problems, and it was uniform across all 4 of the sensors.

 

COMMENTS

A good baseline study that can be used to determine the effectiveness of specific lumbar training exercises, and to also determine if subjects with no pain but having deficits in proprioception, end up with LSI and other spinal conditions.  Once this is determined, use of specific exercises is warranted and justified, especially with the increasing scrutiny of insurers.

 

The surface electrodes were an area of fault in this study, and the authors recognize this fact.  Having specific PT’s choose the subjects for the study always presents the possibility of bias.  It would have been nice if the authors did a better job of describing the neutral they chose, instead of just saying it was between end range flexion and end range extension.  Taking the 15 problematic patients, putting them through an exercise regime, and then re-testing would have been a nice addition to the study.

 

 


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